how to create barcode in asp.net using c# THE LIMBIC LOBES AND THE NEUROLOGY OF EMOTION in Microsoft Office

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THE LIMBIC LOBES AND THE NEUROLOGY OF EMOTION
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from total bilateral temporal lobectomy in adult rhesus monkeys (see also page 397) While these animals were rather placid and lacked the ability to recognize objects visually (they could not distinguish edible from inedible objects), they had a striking tendency to examine everything orally, were unusually alert and responsive to visual stimuli (they touched or mouthed every object within their visual elds), became hypersexual, and increased their food intake This constellation of behavioral changes has been sought in human beings for example, after removal of the temporal lobes but the complete syndrome has been described only infrequently (Marlowe et al; Terzian and Dalle) Pillieri and Poeck have collected cases that have come closest to reproducing the syndrome (Fig 25-3) Unfortunately, many human examples have occurred in conjunction with diffuse diseases (Alzheimer and Pick cerebral atrophies, meningoencephalitis due to toxoplasmosis, herpes simplex, or AIDS) and hence are of less use for anatomic analysis With bitemporal surgical ablations, placidity and enhanced oral behavior were the most frequent consequences; altered sexual behavior and visual agnosia were less frequent In all patients who showed placidity and an amnesic state, the hippocampi and medial parts of the temporal lobe had been destroyed, but not the amygdaloid nuclei Perhaps the most consistent type of reduced emotionality in humans, albeit one that is restricted in scope, is that associated with acute lesions (usually infarcts or hemorrhages) in the right or nondominant parietal lobe Not only is the patient indifferent to the paralysis but, as Bear points out, he is unconcerned about his other diseases as well as personal and family problems, is less able to interpret the emotional facial expressions of others, and is inattentive in general Dimond and coworkers interpret this to mean that the right hemisphere is more involved in affective-emotional experience than the left, which is committed to language Observations derived from the study of split-brain patients and from selective anesthetization of the cerebral hemispheres by intracarotid injection of amobarbital (Wada test) lend some support to this probably oversimpli ed view Rarely, lesions of the left (dominant) hemisphere appear to induce the opposite effect a frenzied excitement lasting for days or weeks The full range of placidity reactions in neurology has not been catalogued Unfortunately, neurologists and psychiatrists have tended to neglect this aspect of behavior
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evokes feelings of pleasure and lust Also, Gorman and Cummings have described two patients who became sexually disinhibited after a shunt catheter had perforated the dorsal septal region This is in keeping with the experience of Heath and Fitzjarrell, who found that infusion of acetylcholine into the septal region (an experimental treatment for Parkinson disease) produced euphoria and orgasm, and with Heath s recordings from the septum of patients during sexual intercourse, showing greatly increased activity with spikes and slow waves Perhaps these are examples of a true overdrive of libido, as contrasted with simple disinhibition of sexual behavior However, we know of no case in which a stable lesion that caused abnormal sexual behavior has been studied carefully by sections of the critical parts of the brain In clinical practice, the commonest cause of disinhibited sexual behavior, next to the aftermaths of head injury and cerebral hemorrhage, is the use of dopaminergic drugs in Parkinson disease Mania may do the same Hyposexuality, meaning loss of libido, is most often due to a depressive illness However, certain medications notably antihypertensive, anticonvulsant, serotoninergic antidepressant, and neuroleptic drugs may be responsible in individual patients A variety of cerebral diseases may also have this effect, in parallel with a loss of interest and drive in a number of spheres Lesions that involve the tuberoinfundibular region of the hypothalamus are known to cause speci c disturbances in sexual function If such lesions are acquired early in life, pubertal changes are prevented from occurring; or, hamartomas of the hypothalamus, as in von Recklinghausen neuro bromatosis and tuberous sclerosis, may cause sexual precocity Autonomic neuropathies and lesions involving the sacral parts of the parasympathetic system, the commonest being prostatectomy, may abolish normal sexual performance but do not alter libido or orgasm Blumer and Walker have reviewed the literature on the association of epilepsy and abnormal sexual behavior They note that sexual arousal as an ictal phenomenon is apt to occur in relation to temporal lobe seizures, particularly when the discharging focus is in the mediotemporal region These authors also cite the high incidence of global hyposexuality in patients with temporal lobe epilepsy Temporal lobectomy in such patients has sometimes been followed by a period of hypersexuality
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