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GROWTH AND DEVELOPMENT OF THE NERVOUS SYSTEM AND THE NEUROLOGY OF AGING
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ROSENBERGER PB: Morphological cerebral asymmetries and dyslexia, in Pavlidis GT (ed): Perspectives on Dyslexia Vol 1 New York, Wiley, 1990, pp 93 107 ROSENBERGER PB: Learning disorders, in Berg B (ed): Principles of Child Neurology New York, McGraw-Hill, 1996, pp 335 369 RUMSEY JM, DONOHUE BC, BRADY DR, et al: A magnetic resonance imaging study of planum temporale asymmetry in men with developmental dyslexia Arch Neurol 54:1481, 1997 RUTTER M, GRAHAM P, YULE W: Clinics in Developmental Medicine Nos 35 and 36 A Neuropsychiatric Study in Childhood London, Heinemann, 1970 RUTTER M, MARTIN JAM (eds): Clinics in Developmental Medicine No 43 The Child with Delayed Speech London, Heinemann, 1972, pp 48 51 SANDAK R, FIEZ JA: Stuttering: A view from neuroimaging Lancet 356: 445, 2000 SCARR S, WEBBER RA, CUTTING MA: Personality resemblance among adolescents and their parents in biologically related and adoptive families J Pers Soc Psychol 40:885, 1981 SCHAIN RJ: Neurology of Childhood Learning Disorders, 2nd ed Baltimore, Williams & Wilkins, 1977 SHAYWITZ SE: Dyslexia N Engl J Med 338:307, 1998 SHAYWITZ SE, SHAYWITZ BA, FLETCHER JM, et al: Prevalence of reading disabilities in boys and girls JAMA 264:998, 1990 SIMOS PG, FLETCHER JM, BERGMAN G, et al: Dyslexia-speci c brain deterioration pro le becomes normal following remedial training Neurology 58:1203, 2002
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SPEARMAN C: Psychology Down the Ages London, Macmillan, 1937 STAPLES R: Responses of infants to color J Exp Psychol 15:119, 1932 STEIN JF, RICHARDSON AJ, FOWLER MS: Monocular occlusion can improve binocular control and reading in dyslexics Brain 123:164, 2000 SWAAB DF, HOFMAN MA: An enlarged suprachiasmatic nucleus in homosexual men Brain Res 537:141, 1990 THURSTONE LL: The Vectors of the Mind Chicago, University of Chicago Press, 1953 TIROSH E: Fine motor de cit: An etiologically distinct entity Pediatr Neurol 10:213, 1994 TRAVIS LE: Speech Therapy New York, Appleton-Century, 1931 TURNER G, TURNER B, COLLINS E: X-linked mental retardation without physical abnormality: Renpenning s syndrome Dev Med Child Neurol 13:71, 1971 WEISS G, HECHTMAN L, MILROY T, PERLMAN T: Psychiatric status of hyperactives as adults: A controlled prospective 15-year follow-up of 63 hyperactive children J Am Acad Child Adolesc Psychiatry 24:211, 1985 WORSTER-DROUGHT C: Congenital suprabulbar paresis J Laryngol Otol 70:453, 1956 YAKOVLEV PI, LECOURS AR: The myelogenetic cycles of regional maturation of the brain, in Minkowski A (ed): Regional Development of the Brain in Early Life Oxford, Blackwell, 1967, pp 3 70 ZAMETKIN AJ, ERNST M: Problems in the management of attention-de cit hyperactivity disorder N Engl J Med 340:40, 1999 ZAMETKIN AJ, NORDAHL TE, GROSS M, et al: Cerebral glucose metabolism in adults with hyperactivity of childhood onset N Engl J Med 323: 1361, 1990
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THE NEUROLOGY OF AGING
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As indicated in the preceding chapter, standards of growth, development, and maturation provide a frame of reference against which every pathologic process in early life must be viewed It has been less appreciated, however, that at the other end of the life cycle, neurologic de cits must be judged in a similar way, against a background of normal aging changes (senescence) The earliest of these changes begins long before the acknowledged period of senescence and continues throughout the remainder of life Not a few medical scientists and physicians believe that all changes in senescence are but the cumulative effects of injury and disease Most authors use the terms aging and senescence interchangeably, but some draw a ne semantic distinction between the purely passive and chronologic process of aging and the composite of bodily changes that characterize this process (senescence) Biologists have measured many of these changes Estimates of the structural and functional decline that accompanies aging from 30 to 80 years are given in Table 29-1 It appears that all structures and functions share in the aging process Some persons withstand the onslaught of aging far better than others, and this constitutional resistance to the effects of aging seems to be familial It can also be said that such changes are unrelated to Alzheimer disease and other degenerative diseases but that in general the changes of aging reduce the ability of the organism to recover from virtually any illness or trauma
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idle, obstinate, and given to reminiscing and as having tremulous hands, quavering voices, stooped posture, and slow, shuf ing steps In so doing, they have selected some of the most obvious effects of aging on the nervous system The lay observer, as well as the medical one, often speaks glibly of the changes of advanced age as a kind of second childhood Old men are boys again, said Aristophanes This view of old age derives from the few resemblances, super cial at best, of the senile dement and the helpless young child
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