HYPODERMIS in .NET framework

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HYPODERMIS
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The hypodermis, or subcutis, located beneath the dermis, is composed mostly of fat, which is an important energy source for the body This layer also contains collagen types I, III, and V As humans age, some of the subcutaneous fat is lost or redistributed into undesired areas This phenomenon contributes to the aged appearance Fat injections have been employed to move fat from undesired areas into desired areas where fat has been lost, such as the lower face (see 23) The adipocytes secrete a hormone called leptin, a product of the obesity (ob) gene Leptin exhibits a regulatory effect on human metabolism and appetite and therefore affects adipose tissue mass Leptin has been shown to be higher in the serum of obese patients, with commensurate levels found in body fat percentage59 It is believed that a higher percentage of body fat results in elevated leptin levels and the turning off of signals to the brain for appetite reduction Recombinant leptin injections in mice have been associated with reduction of weight and body fat percentage60 However, more research is needed to ascertain the therapeutic potential of leptin in humans
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Although the epidermis is the target of most topical cosmetic products because most do not penetrate to the dermis, the dermis is the target for many of the injectable treatments for aging The dermis is an extremely important component in skin appearance because it is responsible for imparting thickness and suppleness to the skin A thinner dermis and an altered DEJ are hallmarks of aged skin Loss of collagen, elastin, and GAGs located primarily in the dermis contribute significantly to cutaneous aging Various measures intended to prevent or retard aging target these key constituents of the dermis
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1 Monnier VM, Kohn RR, Cerami A Accelerated age-related browning of human collagen in diabetes mellitus Proc Natl Acad Sci U S A 1984;81:583
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2 Schnider SL, Kohn RR Glucosylation of human collagen in aging and diabetes mellitus J Clin Invest 1980;66:1179 3 Schnider SL, Kohn RR Effects of age and diabetes mellitus on the solubility and nonenzymatic glucosylation of human skin collagen J Clin Invest 1981;67:1630 4 Yamada K, Miyahara Y, Hamaguchi K, et al Immunohistochemical study of human advanced glycosylation endproducts (AGE) in chronic renal failure Clin Nephrol 1994;42:354 5 Thornalley PJ Advanced glycation end products in renal failure J Ren Nutr 2006;16:178 6 Vitek MP, Bhattacharya K, Glendening JM, et al Advanced glycation end products contribute to amyloidosis in Alzheimer disease Proc Natl Acad Sci U S A 1994;91:4766 7 Yan SD, Chen X, Schmidt AM, et al Glycated tau protein in Alzheimer disease: a mechanism for induction of oxidant stress Proc Natl Acad Sci U S A 1994;91:7787 8 Takeuchi M, Kikuchi S, Sasaki N, et al Involvement of advanced glycation endproducts (AGEs) in Alzheimer s disease Curr Alzheimer Res 2004;1:39 9 Dyer DG, Dunn JA, Thorpe SR, et al Accumulation of Maillard reaction products in skin collagen in diabetes and aging J Clin Invest 1993;91:2463 10 Verzijl N, DeGroot J, Odehinkel E, et al Age-related accumulation of Maillard reaction products in human articular cartilage collagen Biochem J 2000;350: 381 11 Mizutari K, Ono T, Ikeda K, et al Photoenhanced modification of human skin elastin in actinic elastosis by N(epsilon)(carboxymethyl)lysine, one of the glycoxidation products of the Maillard reaction J Invest Dermatol 1997;108:797 12 Tomasek JJ, Haaksma CJ, Eddy RJ, et al Fibroblast contraction occurs on release of tension in attached collagen lattices: dependency on an organized actin cytoskeleton and serum Anat Rec 1992; 232:359 13 Howard EW, Benton R, Ahern-Moore J, et al Cellular contraction of collagen lattices is inhibited by nonenzymatic glycation Exp Cell Res 1996;228:132 14 Rittie L, Berton A, Monboisse JC, et al Decreased contraction of glycated collagen lattices coincides with impaired matrix metalloproteinase production Biochem Biophys Res Commun 1999;264:488 15 Wondrak GT, Roberts MJ, Jacobson MK, et al Photosensitized growth inhibition of cultured human skin cells: mechanism and suppression of oxidative stress from solar irradiation of glycated proteins J Invest Dermatol 2002;119:489 16 Nelson B, Majmudar G, Griffiths C, et al Clinical improvement following dermabrasion of photoaged skin correlates with synthesis of collagen I Arch Derm 1994;130:1136 17 Oikarinen A The aging of skin: chronoaging versus photoaging Photodermatol Photomed 1990;7:3 18 Craven NM, Watson RE, Jones CJ, et al Clinical features of photodamaged human skin are associated with a reduction in collagen VII Br J Derm 1997;137: 344 19 Vrhovski B, Weiss AS Biochemistry of tropoelastin Eur J Biochem 1998;258:1
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