COSMETIC DERMATOLOGY: PRINCIPLES AND PRACTICE in .NET

Generating QR-Code in .NET COSMETIC DERMATOLOGY: PRINCIPLES AND PRACTICE

COSMETIC DERMATOLOGY: PRINCIPLES AND PRACTICE
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50 Mizuno H, Zuk PA, Zhu M, et al Myogenic differentiation by human processed lipoaspirate cells Plast Reconstr Surg 2002;109:199 51 De Ugarte DA, Morizono K, Elbarbary A, et al Comparison of multi-lineage cells from human adipose tissue and bone marrow Cells Tissues Organs 2003;174: 101
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52 Kokai LE, Rubin JP, Marra KG The potential of adipose-derived adult stem cells as a source of neuronal progenitor cells Plast Reconstr Surg 2005;116:1453 53 Draelos ZD, Marenus KD Cellulite Etiology and purported treatment Dermatol Surg 1997;23:1177 54 Draelos ZD The disease of cellulite J Cosmet Dermatol 2005;4:221
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55 Pi rard GE Cellulite: from standing fat herniation to hypodermal stretch marks Am J Dermatopathol 2000;22:34 56 Alexiades-Armenakas M Laser and lightbased treatment of cellulite J Drugs Dermatol 2007;6:83 57 Hexsel DM, Mazzuco R Subcision: a treatment for cellulite Int J Dermatol 2000;39:539
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CHAPTER 3 FAT AND THE SUBCUTANEOUS LAYER 21
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CHAPTER 4 Immunology of the Skin
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Little is known about the relationship between immunology and skin appearance; however, it is certain that the immune system plays an important role in the health of the skin Work is ongoing to help elucidate how this vital system interacts with the largest organ of the body It is very likely that this segment of research, as it pertains to the cosmetic dermatology arena, will offer significant potential for discovery of new therapeutics and procedures in the next several years This chapter will serve as a brief introduction to the skin as an immune organ and how the immune response plays a role in cosmetic dermatology In the past, the skin was viewed primarily as a barrier mechanism to prevent invading pathogens and other environmental toxins, including UV radiation, from penetrating into internal organs However, we now know that the skin essentially acts as an immense and integral immune organ and first point of contact with the environment, capable of initiating an intricate series of events leading to host defense A basic review of skin immunology, including the role of cytokines and growth factors, will be provided as an important part of this discussion Mechanisms of various immune responses found in skin disease, the interplay between innate immunity and extracellular matrix synthesis, as well as emerging immunebased treatments will also be highlighted Finally, the relevance of the local immune system and its relationship to skin aging, particularly photoaging, will be briefly reviewed response, on the other hand, occurs slowly and activation of adaptive immune cells, such as B and T cells, requires that receptors undergo gene rearrangements The adaptive immune system can mount either humoral immunity (B cells, which make antibodies) or cell-mediated immunity (T cells) Furthermore, the adaptive immune system is also responsible for immune memory, which confers long-term protection to the host Although the two systems appear distinct, they are not separate, and in fact can act synergistically, insofar as the innate immune system instructs the adaptive immune response and the adaptive immune system influences the innate system In the epidermis, the two main innate cells are the keratinocytes and Langerhans cells In addition, neutrophils, macrophages, and dendritic cells present within the dermis also play a role in innate immunity When a foreign substance is encountered, activation of innate cells occurs through PRRs, including the Tolllike receptors (TLRs), which are reviewed below Upon activation, the innate cells become capable of inducing a direct antimicrobial response by producing factors that can help protect the host from external insults These factors include reactive oxygen and nitrogen intermediates (also known as free radicals ) and antimicrobial peptides In addition, activated innate cells produce cytokines and other inflammatory mediators that can instruct adaptive immunity Paradoxically, the same innate immune response can induce proinflammatory cytokine production that can lead to inflammation and tissue injury, thereby facilitating disease pathology
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