COSMETIC DERMATOLOGY: PRINCIPLES AND PRACTICE in VS .NET

Creation Quick Response Code in VS .NET COSMETIC DERMATOLOGY: PRINCIPLES AND PRACTICE

COSMETIC DERMATOLOGY: PRINCIPLES AND PRACTICE
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ANTIOXIDANT THEORY
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The body has developed defense mechanisms, known as antioxidants, which protect against the ravages of free radicals by reducing and neutralizing them Antioxidative enzymes that naturallyoccur in the skin include superoxide dismutase, catalase, and glutathione peroxidase (GPX); the nonenzymatic endogenous antioxidative molecules are alpha-tocopherol (vitamin E), ascorbic acid (vitamin C), glutathione, and ubiquinone12 As part of the natural aging process, our defense mechanisms diminsh, while the production of ROS increases This leads to an imbalance and an elevated number of unchecked free radicals that damage DNA, cytoskeletal elements, cellular proteins, and cellular membranes Moreover, many of the body s antioxidant defense mechanisms are inhibited by UV and visible light13,14 Additionally, UV light exposure is known to cause an increase in free radical formation15 Researchers are currently studying the potential of using exogenous antioxidants to affect this process and mitigate the damage caused by free radicals Many believe that topical application and oral consumption of combinations of antioxidants may result in a sustained antioxidant capacity of the skin because of synergistic actions of the combined antioxidants Antioxidants may be particularly useful in UVAinduced skin alterations that are believed to be determined, in large part, by oxidative processes In fact, topical application of antioxidants has been shown to raise
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inactive before reaching the target Stabilizing ingredients in formulation and packaging them in order to minimize air and light exposure are challenging tasks Absorption is also important and depends on several factors such as the molecular form of the compound, its pH, whether it is water soluble or fat soluble, and the vehicle that contains the product This chapter will include a discussion of the most popular types of antioxidants found in cosmetic products or taken orally by cosmetic patients
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Vitamin E
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Found in vegetables, oils, seeds, nuts, corn, soy, whole wheat flour, margarine, and in some meat and dairy products, vitamin E, or tocopherol, is actually a universal term for a group of compounds comprising of tocol and tocotrienol derivatives, specifically four pairs of racemic stereoisomers (Fig 34-1) Of the four tocopherols (alpha-, beta-, gamma-, and delta-), alpha-tocopherol (also known as AT) has the highest activity Since the discovery that vitamin E is the primary lipid-soluble antioxidant in skin that protects cells from oxidative stress,18 practitioners have used it to treat a wide variety of skin lesions The general public has frequently looked to it for treatment of minor burns, surgical scars, and other wounds, even though its use for dermatoses has not been approved by the FDA In addition, vitamin E is touted as beneficial in protecting against cardiovascular disease because lipid peroxidation is a major contributor to atherosclerosis and cardiovascular disease Administration of vitamin E is thought to decrease the extent of lipid peroxidation and protect against cardiovascular disease19 In the same way that vitamin E protects the cells from lipid peroxidation in the arteries, it protects the cell membranes in the skin from peroxidation Authors have shown a correlation between dietary deficiency of vitamin E and an increase in oxidative stress and cell injury18 In 1993, Tanaka et al reported that ROS induce changes in the biosynthesis of collagen and glycosaminoglycans (GAGs) in cultured human dermal fibroblasts20 This alteration was prevented with the addition of alpha-tocopherol to the fibroblasts In addition, vitamin E has been shown to lower prostaglandin E2 production,21 and augment interleukin-2 (IL-2) production, leading to anti-inflammatory and immunostimulatory activity It is believed that this stabilization effect may play a role in collagen biosynthesis22 Thus far, in vivo attempts to measure and link changes in collagen production with altered concentrations of vitamin E have proved inconclusive A lower incidence of infectious disease and cancer has been observed in
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