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them down. If these genes become mutated from carcinogen exposure or if you happen to be born with mutated forms cancer may result.
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Each oncogene or tumor suppressor gene, of which there are one hundred known variations, has two alleles; one from each parent. Generally, a mutation first occurs in one of the alleles or it is inherited from one of the parents.
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Some carcinogens, such as aflatoxin, require only low exposure for a short time to cause cancer. Other carcinogens, such as benzene, are relatively weaker and require higher doses for longer periods of time. People with genetically inherited high levels of detoxification enzymes obviously deal with relatively more carcinogens than those with genetically inherited lower levels. The same can be said for our DNA repair capability and immune systems, the next lines of defense: the better they function, the more they protect us. Age is also a very important factor. Animal studies have shown that because of higher levels of cell growth and turnover, younger animals are more susceptible to cancer induction than older animals, where cell growth and turnover is lower. Humans behave the same way. Cancer development has three stages: initiation, promotion, and progression. Tumor initiation begins when DNA in skin cells, for example, is damaged by exposure to a carcinogen, such as ultraviolet light or a PAH, an event that would not likely occur in the presence of strong detoxification capabilities. However, if DNA damage does occur and it is not repaired, it can lead to a genetic mutation in either a tumor suppressor gene or an oncogene. Metaphorically speaking, the brakes fail or the accelerator sticks or both. Either way, cell proliferation increases and cancer looms. In many cancers, both oncogenes and tumor suppressor genes are mutated. The induction of skin cancer in experimental animals provides a good illustration of the three stages of tumor formation. Over the life span of an animal, a mutation can be produced in an oncogene in skin cells from low dosages of a potent carcinogen without giving rise to cancer. However, if the animal with the mutated but dormant oncogene is exposed for a long period of time to a tumor promoter, cancer will develop. It doesn t matter if the tumor promoter is given right after the
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mutation occurs or months later. This example of the induction of cancer in experimental animals, in which tumor initiation is followed sequentially by tumor promotion and progression, is very important not only for the understanding of the development of cancer but also for a rational and effective prevention of cancer. Similar sequential stages are also believed to be involved in human cancer. Estrogens and androgens can act as tumor promoters by stimulating mutated cells to grow and multiply, which can lead to breast or prostate cancer. Further stimulation with low doses of other environmental toxins and carcinogens can cause the number of initiated or mutated cells, called precancerous cells, to expand and progress to true cancer cells, which can then invade surrounding tissues and metastasize.
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Detoxification Strategies for Cancer
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The emphasis of this book on detoxification is to prevent diseases like cancer. But that doesn t mean we scrap a detoxification strategy if cancer is diagnosed. In fact, once the stage of cancer formation is identified, it is possible, and necessary, to develop a customized intervention strategy with the appropriate protective compounds. The chart on page 129 depicts the various stages of the development of cancer and strategies that have been found to be effective in each stage. The formulation of essential fats is also very important in cancer prevention. The sulfur compounds from cruciferous vegetables and garlic, the flavonoid compounds from fruits and vegetables, the terpenoids and curcuminoids from spices, chlorophyllin from green vegetables, and D-glucarate from fruits and vegetables are particularly potent inhibitors of the tumor initiation stage because they enhance Phase I and Phase II detoxification and DNA repair. In terms of tumor promotion and progression, the antioxidants from fruits and vegetables and critical cellular antioxidants plus essential fats and immune enhancement compounds are very effective because of inhibiting free radicals, inflammation, and cell proliferation, as well as stimulating immune function and cancer cell death. These compounds listed in the Breakthrough Supplements for Cancer table on page 130 are potent inhibitors of the induction of cancer because they stimulate detoxification function. They show promise as agents for the treatment of cancer progression. Many studies have found that they inhibit the proliferation of cancer cells, stimulate apoptosis or cell death, stimulate normal differentiation in cancer cells, counteract the growth of blood vessels to tumors (antiangiogenesis),
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